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E-brochureMedicine (Neurology, Rheumatology, Clinical Immunology)

Master's Program Human Sciences, Graduate School of Medicine
Doctor's Program Medical Science Division, Department of Medical Science, Graduate School of Medicine, Science and Technology

Staff List

Professor SEKIJIMA Yoshiki
Associate Professor SHIMOJIMA Yasuhiro
Senior Assistant Professor KATO Nagaaki, KODAIRA Minori, MIYAZAKI Daigo, HINENO Akiyo
Assistant Professor KISHIDA Dai, YOSHINAGA Tsuneaki

Contact

E-mail : sannai(at)shinshu-u.ac.jp
※ Change (at) to @ when you send an e-mail.

Summary of Activity

Department of Medicine (Neurology and Rheumatology) is focusing on the development of skills to explore the patients' whole body.
In the Neurology area, our target is total nervous system of the whole body which consist of the brain, spine, peripheral nerve, and muscles. There are many neurological disorders which do not show any abnormality in blood tests or imaging examination. Therefore it is very important to make a correct diagnosis through the whole body physical examinations and neurological examinations.
In the Rheumatology area, we see the patients with connective tissue disorders which mainly involve the whole body joints. You might be able to get the ability to find abnormal joints by touching and feeling the patients' joints by your own hands.
Amyloidosis is a disorder characterized by the deposition of abnormal protein called 'amyloid' into the whole body, such as the brain, heart, kidney, liver, digestive tract, peripheral nervous system, muscles, and endocrine systems. These visceral organs are also involved among the patients with rheumatic diseases.
Therefore, you may get the specialized skills as an neurologist and rheumatologist and also get the general skills to see and manage the patients' whole body issue. See following pages to check the details of our research works.

Divisions:
1. Amyloidosis team
2. Brain Disease Research team
3. Muscular disorder team
4. Rheumatic disorder team

Major Publications

1. Nakagawa M, Sekijima Y, Tojo K, Ikeda S. High prevalence of ATTR amyloidosis in endomyocardial biopsy-proven cardiac amyloidosis patients. Amyloid 2013;20:138-140.

2. Tachibana N, Kinoshita M, Saito Y, Ikeda S. Identification of the N-methyl-D   -aspartate receptor (NMDAR)-related epitope, NR2B, in the normal human ovary: implication for the pathogenesis of anti-NMDAR encephalitis. Tohoku J Exp Med 2013;230:13-16. 
 
3. Naito K, Fukushima K, Suzuki S, Kuwahara M, Morita H, Kusunoki S, Ikeda S. Intravenous immunoglobulin (IVIg) with methylpredonisolone pulse therapy for motor impairment of neuralgic amyotrophy: clinical observations in 10 cases. Intern Med 2012;51:1493-1500.

4. Ikeda S, Sekijima Y, Tojo K, Koyama J. Diagnostic value of abdominal wall fat biopsy in senile systemic amyloidosis. Amyloid 2011;18:211-215.

5. Sekijima Y, Uchiyama S, Tojo K, Sano K, Shimizu Y, Imaeda T, Hoshii Y, Kato H, Ikeda S. High prevalence of wild-type transthyretin deposition in patients with idiopathic carpal tunnel syndrome: a common cause of carpal tunnel syndrome in the elderly. Hum Pathol 2011;42:1785-1791.

6. Kodaira M, Morita H, Shimojima Y. Ikeda S. Electrophysiological features of familial amyloid polyneuropathy. Amyloid 2011;18:10-18.

7. Tsuchiya-Suzuki A, Yazaki M, Kametani F, Sekijima Y, Ikeda SI. Wild-type trans-thyretin significantly contributes to the formation of amyloid fibrils in familial amyloid polyneuropathy with amyloidogenic transthyretin Val30Met. Hum Pathol 2011;42:236-243.

8. Ikeda S, Sekijima Y, Tojo K, Koyama J. Diagnostic value of abdominal wall fat biopsy in senile systemic amyloidosis. Amyloid 18: 211-215, 2011.

9.Tsuchiya-Suzuki A, Yazaki M, Kametani F, Sekijima Y, Ikeda S. Wild-type transthyretin significantly contributes to the formation of amyloid fibrils in familial amyloid polyneuropathy patients with amyloidogenic transthyretin Val30Met. Hum Pathol 42:236-243, 2011.

10. Kodaira M, Morita H, Shimojima Y, Ikeda S. Electrophysiological features of familial amyloid polyneuropathy in endemic area. Amyloid 18:10-18, 2011.


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