Graig Crandall, Ph.D.
Associate Professor of Internal Medicine at the
Dr.
Crandall received a doctorate in Physiology from the University of North Texas Health Science Center at
Heat stress results in significant cardiovascular
adjustments as evidenced by pronounced increases in heart rate, cardiac
contractility, sympathetic activity, and reductions in renal and splanchnic
vascular conductance. In humans
these events are accompanied by marked reductions in orthostatic
tolerance. Mechanisms by which heat
stress reduces orthostatic tolerance remain unclear. Possible mechanisms that will be
addressed include altered baroreflex responsiveness as well as attenuated
vasoconstriction to adrenergic agents.